David Mattson Jr, MD

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Assistant Professor, Department of Radiation Medicine
Director, Breast Radiation Oncology Program
Roswell Park Cancer Institute
Clinical Assistant Professor of Radiation Oncology
School of Medicine and Biomedical Sciences, State University of New York at Buffalo

Profile

Areas of Expertise: 
Breast cancer

Research

Research Interests: 
Partial breast irradiation and hypofractionated partial breast irradiation

Background Information

Education and Training: 
MD - John A. Burns School of Medicine, University of Hawaii at Manoa, Honolulu, HI
Residency: 
University Hospital, State University of New York Health Sciences Center, Syracuse, NY
University of Iowa Hospital & Clinics, Iowa City, IA
Fellowships: 
University of Iowa Hospital & Clinics, Iowa City, IA
National Cancer Institute, Bethesda, MD

Publications

Publications: 
  • Mattson DM, Dornfeld KJ, Spitz DR, Orcutt KP, Li L, Aykin-Burns N, Parsons AD, Dayal D, Ahmad IM, Simons AL. Cisplatin combined with zidovudine enhances cytotoxicity and oxidative stress in human head and neck cancer cells via a thiol-dependent mechanism. Free radical biology and medicine 2009; 46(2):232-237
  • Scarbrough PM, Mattson D, Gius D, Watson WH, Spitz DR. Inhibitors of glucose and hydroperoxide metabolism potentiate 17-AAG-induced cancer cell killing via oxidative stress. Free radical biology and medicine 2010; 49(Suppl. 1):S69
  • Wilson JP , Mattson D , Edge SB. Is there a need for axillary dissection in breast cancer? . Journal of the National Comprehensive Cancer Network : JNCCN 2011; 9(2):225-230
  • Scarbrough PM, Mattson DMK, Aykin-Burns N, McCormick ML, Gius D, Spitz DR. Inhibitors of Glucose and Hydroperoxide Metabolism Enhance 17-allylamino-17-demethoxygeldanamycin Toxicity in Human Breast Cancer Cells. Free radical biology and medicine 2009; 47(Suppl. 1):S174
  • Scarbrough PM, Mapuskar KA, Mattson DM, Gius D, Watson WH, Spitz DR. Simultaneous inhibition of glutathione- and thioredoxin-dependent metabolism is necessary to potentiate 17AAG-induced cancer cell killing via oxidative stress. Free radical biology and medicine 2012; 52(2):436-443